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Figure 1.

Pedigree, haplotype analysis, and mutation analysis of the families in this study.

A: Pedigree and disease-haplotype segregation of family A. Blackened symbols represent affected individuals with an abnormal limb phenotype. White symbols represent individuals with a normal limb phenotype. Circles and squares indicate females and males, respectively. The arrows identify the proband and the disease-haplotype is boxed. B: Pedigree and disease-haplotype segregation of family B. C: HOXD13 missense mutations in the two families.

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Figure 2.

Photographs and radiographs of individuals.

Phenotypic spectrum of the two families showing typical SD1-c bilateral 3/4 fingers syndactyly (A, B, H, I and J) and unilateral 3/4 fingers syndactyly (G, M and N); transformative SD1-c bilateral 3/4 finger syndactyly with right fifth finger clinodactyly (K and L); mild SPD, bilateral 3/4 fingers syndactyly with phalanx duplication (E and F) and unilateral 3/4 fingers syndactyly with 4/5 fingers synpolydactyly (O and P). Other phenotypes included isolated unilateral partial webbing of 2/3 toes like SD1-a (D) and isolated unilateral adduction deformity of toes (C).

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Table 1.

The clinical spectrum in family A and family B with SD1-c.

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Table 2.

Two-point LOD score of chromosome 2q markers at various recombination fractions.

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Figure 3.

Schematic presentation of the HOXD13 structure and sequences alignment analysis.

A: Schematic presentation of the HOXD13 structure and annotated mutations identified in families. B: Partial amino acid sequences alignment in homeodomain of HOXD13 among several species. The position of mutated amino acid of our cases is indicated by the black box.

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Table 3.

Overview of annotated HOXD13 mutations and clinical observations in family members or sporadic cases.

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Figure 4.

Transcriptional activity of wild-type and mutant HOXD13 proteins at the human EPH7A promoter.

The R31Q (p.R306Q), R31G (p.R306G) and I47L (p.I322L) mutants show a significantly diminished stimulation compared with the wild-type control at approximately 62%, 60% and 58% of the wild-type HOXD13, respectively. Bars represent firefly/Renilla luciferase ratios for the different constructs.

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