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Figure 1.

The location of cerebral microbleeds (CMBs) on 3D-enhanced T2*-weighted gradient echo sequence of MRI (arrows).

(A) CMBs in frontal lobe. (B) CMBs in thalamus.

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Figure 1 Expand

Figure 2.

White matter hyperintensities on T2-fluid-attenuated inversion recovery sequence (T2-FLAIR).

Deep white matter hyperintensities (DWMH) and peri-ventricular hyperintensities (PVH) was indicated by arrow and arrow head, respectively.

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Figure 2 Expand

Table 1.

Clinical Characteristics.

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Table 1 Expand

Table 2.

Risk Factors for CMBs, PVH, DWMH and LI (OR, 95% CI).

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Table 2 Expand

Figure 3.

The relationship between cerebral microbleeds (CMBs) and white matter hyperintensities.

The proportion of patients with CMBs increased with the severity of deep white matter hyperintensities (DWMH) (p<0.001, χ2 test) and peri-ventricular hyperintensities (PVH) (p<0.001, χ2 test). Each bar represents the number of patients.

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Figure 3 Expand

Table 3.

Correlations amongst CMBs, WML, LI and risk factors (r).

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Table 3 Expand

Table 4.

Correlation between the presence of CMBs and PVH, DWMH stratified by hypertension(r).

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Table 4 Expand

Table 5.

Correlation between the distribution of CMBs and presence of PVH, DWMH (%, n).

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Table 5 Expand

Table 6.

Correlation between the severity of CMBs and presence of PVH, DWMH (%, n).

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Table 6 Expand