Table 1.
Subject demographics.
Figure 1.
Association between liver biochemical test parameters and serum glucose.
Tertiles of plasma LnALT, LnAST, LnGGT and serum LnTriglyceride (TG) (mmol/L) in association with nonfasting serum levels (mmol/L) of glucose. Asterisks (*p<0.05, **p<0.01, ***p<0.001) represents significant difference between groups using linear regression analysis, correcting for relation to sibling relationship, age, gender, smoking, alcohol use in g/day, and number of hepatotoxic medication. NS: not significant.
Table 2.
Nonfasting liver enzymes and triglycerides in offspring of long-lived siblings and control subjects.
Table 3.
Fasting liver enzymes and triglycerides in offspring of long-lived siblings and control subjects.
Figure 2.
Unenhanced Computed Tomography (CT) scan of the upper abdomen, axial view.
Normal liver in a 58-year-old female; liver attenuation is 62 HU, spleen is 55 HU, liver-spleen ratio is 1.1 (a). 64-year-old female with liver steatosis; the liver parenchyma (28 HU) is relatively hypodense compared to the spleen (55 HU), liver-spleen ratio is 0.5 (b). HU: hounsfield units.
Table 4.
Computed tomography markers of liver steatosis in offspring of long-lived siblings and control subjects.