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Table 1.

Composition and characterization of non-loaded liposomal formulations.

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Table 1 Expand

Figure 1.

Characterization of liposomal formulations.

(A) Representative Cryo-TEM image of DLPC/Chol/Cholesteryl/PEG600-Chol (5∶3.5∶1∶0.5) liposomes extruded through a 200 nm pore size membrane. (B) Confocal fluorescence image of a single liposome tagged on its lipid bilayer with Marina Blue-DHPE (blue) and its corresponding fluorescence intensity profile. (C) Confocal fluorescence image of a single Marina Blue-labeled liposome containing AlexaFluor594-labeled LPS (red) and their corresponding fluorescence intensity profiles. (D) Confocal fluorescence image of a single Marina Blue-labeled liposome containing fluorescein-labeled poly (I:C) and their corresponding fluorescence intensity profiles. (E) Schematic representation of the liposomal IS-cocktail (NLc) showing the presence of both encapsulated LPS (red) and poly (I:C) (green) in the lipidic bilayer of liposomes. (F) Confocal fluorescence image of a single liposome containing both fluorescein-labeled poly (I:C) (green) and AlexaFluor594-labeled LPS (red) and their corresponding fluorescence intensity profiles.

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Table 2.

Efficiencies for the encapsulation of LPS and poly (I:C).

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Figure 2.

Cytotoxicity of NL2, LPS, NL2, poly (I:C), and NLc formulations in ZFL cells by MTT-based assay.

(A) Viability of ZFL after 24 h incubation with liposome-encapsulated LPS (NL2, LPS, green bars) at Dose 1 = 1 mg/ml liposome with 50 µg/ml LPS, Dose 2 = 0.5 mg/ml liposome with 25 µg/ml LPS and Dose 3 = 0.20 mg/ml liposome with 10 µg/ml LPS. The white bar is the empty liposome control (NL2,n, 1 mg/ml liposome) and the blue bar is the free LPS control (50 µg/ml). (B) Viability of ZFL after 24 h incubation the liposome-encapsulated poly (I:C) (NL2, poly (I:C), green bars) at Dose 1 = 1.5 mg/ml liposome with 50 µg/ml poly (I:C), Dose 2 = 0.75 mg/ml liposome with 25 µg/ml poly (I:C) and Dose 3 = 0.375 mg/ml liposome with 10 µg/ml poly (I:C). The white bar is the empty liposome control treatment (NL2,n, 1.5 mg/ml liposome) and the red bar is the non-encapsulated poly (I:C) control (50 µg/ml). (C) Viability of ZFL cells after 24 h incubation with liposomal LPS-poly (I:C) cocktail (NLc, green bars) at Dose 1 = 1.5 mg/ml liposome with 50 µg/ml poly (I:C) and 25 µg/ml LPS, Dose 2 = 0.75 mg/ml liposome with 25 µg/ml poly (I:C) and 12.5 µg/ml LPS and Dose 3 = 0.375 mg/ml liposome with 12.5 µg/ml poly (I:C) and 6.25 µg/ml LPS. The white bar is the empty liposome control treatment (NL2,n, 1.5 mg/ml liposome), the blue bar indicates the free LPS (25 µg/ml) and the red bar is the free (I:C) control (50 µg/ml). Non-treated cells were used as 100% viability control (dotted line). Data represent means ± SD of three independent experiments. Differences were analyzed using One-way ANOVA followed by Tukey's post test. **, p<0.01; ***, p<0.001.

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Figure 3.

Endocytosis of NLc formulation by ZFL cells.

(A) Flow cytometry time-course comparison of the membrane-bound (dark grey bar) versus the endocyted liposomes (light grey bar) after incubation with NLc (750 µg/ml liposome, 25 µg/ml poly (I:C) and 12.5 µg/ml LPS) at the indicated times. Data represent means ± SD of three independent experiments. (B) Effect of chemical inhibitors on the endocytosis of the NLc (750 µg/ml liposome, 25 µg/ml poly (I:C) and 12.5 µg/ml LPS). Inhibitors were used at the following concentrations: MβCD at 5 mM, EIPA at 50 µM, sucrose at 300 mM and W at 100 nM. The uptake of cells without inhibitors (NLc bar) was used as 100% uptake control and non-treated cells were used as control (control bar). Data represent means ± SD of three independent experiments. Differences were analyzed using One-way ANOVA followed by Tukey's post test. *, p<0.05; **, p<0.01; ***, p<0.001. (C) Confocal microscopy images of fluorescent liposomes (NLc) endocyted by ZFL cells. Cells were incubated for 30 min, 1.5 h and 16 h with NLc containing DHPE-Fluorescein (green) at a 0.05 molar ratio. Cell membranes were stained with CellMask (red) and the nucleus was stained with Hoechst (blue).

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Figure 4.

Endocytosis of NLc formulation by trout macrophages.

(A) Flow cytometry time-course comparison of the membrane-bound (dark grey bar) versus the endocyted liposomes (light grey bar) after incubation with 750 µg/ml liposome-encapsulated 25 µg/ml poly (I:C) and 12.5 µg/ml LPS at the indicated times. Data represent means ± SD of three independent experiments. (B) Effect of chemical inhibitors on the endocytosis of NLc (750 µg/ml liposome-encapsulated 25 µg/ml poly (I:C) and 12.5 µg/ml LPS) macrophages uptake. Inhibitors were used at the following concentrations: MβCD at 5 mM, EIPA at 50 µM, sucrose at 150 mM and W at 100 nM. The uptake of cells not treated with inhibitors (NLc bar) was used as 100% uptake control and non-treated cells were used as control (control bar). Data represent means ± SD of 3 independent experiments. Differences were analyzed using One-way ANOVA followed by Newman-Keuls post-test. *, p<0.05; **, p<0.01. (C) Confocal microscopy images of fluorescent liposomes (NLc) endocyted by macrophages. Cells incubated 30 min, 1 h and 16 h with NLc containing DHPE-Fluorescein (green) at a 0.05 molar ratio. Cell membranes were stained with CellMask (red) and nucleus with Hoechst (blue).

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Figure 5.

Analysis of gene expression in ZFL cell culture (A) and trout macrophage primary cell culture (B) after 16 h exposure to liposomes.

NL2,n = liposomes without immunostimulants (750 µg/ml), NLc Dose 1 = liposomes (750 µg/ml) containing 25 µg/ml poly (I:C) and 12.5 µg/ml LPS, NLc Dose 2 = liposomes (375 µg/ml) containing 12.5 µg/ml poly (I:C) and 6.25 µg/ml LPS, and LPS+poly (I:C) = stimulation control (25 µg/ml poly (I:C), 12.5 µg/ml LPS). Elongation factor (EF1) was used as reference gene for ZFL cells and 18S for trout macrophages. IFN (φ for ZFL and α for macrophages), GIG2, CCL4, IL-6 and TNFα abundance was analyzed by Q-PCR (left panel) and conventional PCR (right panel). Data represent means ± SD of 3 independent experiments. Values with asterisk are statistically significant relative to the control (*, p<0.05; **, p<0.01; ***, p<0.001) and values with letters (a,b) are statistically significant relative to NLc Dose 1 (a, p<0.001, b, p<0.05). Differences were analyzed using One-way ANOVA and Tukey's post test. (C) TNFα secretion from trout macrophages stimulated with liposomes for 16 h was assessed by Western blot. NLc Dose 2 = 375 µg/ml liposomes, 12.5 µg/ml poly (I:C), 6.25 µg/ml LPS, NL2,n = empty liposomes (375 µg/ml) and LPS = stimulation control (6.25 µg/ml). A representative Western Blot is shown.

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Figure 6.

In vivo NLc formulation toxicities.

Survival of zebrafish embryos was recorded every 24(hpf) (A) and 72 h post-hatching (hph) (B) after exposure to four concentrations of liposomal IS cocktail (red, NLc Dose 1 = 750 µg/ml liposomes, 25 µg/ml poly (I:C) and 12.5 µg/ml LPS; NLc Dose 2 = 1.5 mg/ml liposomes, 50 µg/ml poly (I:C) and 25 µg/ml LPS; NLc Dose 3 = 3 mg/ml liposomes, 100 µg/ml poly (I:C) and 50 µg/ml LPS; NLc Dose 4 = 6 mg/ml liposomes, 200 µg/ml poly (I:C) and 100 µg/ml LPS). Liposomes without encapsulated immunostimulants (grey, NL2,n Dose 2 = 1.5 mg/ml, NL2,n Dose 4 = 6 mg/ml) and non-treated embryos (blue) were used as controls. Non-encapsulated LPS (black, 25 µg/ml and 100 µg/ml) was used as mortality control. Differences were analyzed using log rank test. *, p<0.05; **, p<0.01; ***, p<0.001.

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