Figure 1.
Plasma concentrations of calcium and phosphate in the 3 groups with different amino acid intake.
LAA group: <1.5 g/kg/day of mean AA intake; MAA group: 1.5-2 g/kg/d of mean AA intake; HAA group: >2 g/kg/day of mean AA intake.
Figure 2.
Calcium urine output in the 3 groups with different amino acid intake.
LAA group: <1.5 g/kg/day of mean AA intake; MAA group: 1.5-2 g/kg/d of mean AA intake; HAA group: >2 g/kg/day of mean AA intake.
Figure 3.
Linear regression function showing the relationship between the cumulative deficit of phosphorus intake and the plasma concentrations of calcium and phosphate.
Figure 4.
Hypothesis for the mechanism of the Placental Interrupted Feeding syndrome of the preterm infant (PI-Feeding syndrome).
The abrupt interruption of the continuous placental flow of amino acids and energy promotes catabolism and ion release by the cell. This causes disturbances such as hyperphosphatemia and hyperkalemia, despite the break in phosphorus and potassium supply.
Figure 5.
Hypothesis for the mechanism of the Placental Incompletely Restored Feeding syndrome of the preterm infant (PI-ReFeeding syndrome).
The parenteral supply of amino acids and energy maintains the cell in an anabolic state and promotes its uptake of phosphorus and potassium. This causes a decrease of their plasma concentrations in the absence of an adequate intake.