Table 1.
Characteristics of the patient groups and routine blood analysis.
Table 2.
Correlations between stress hormones and selected inflammatory markers.
Figure 1.
Plasma levels of ACTH and cortisol in CAD patients undergoing PCI.
Blood samples were collected from stable angina pectoris (SAP) and from acute coronary syndrome (ACS) patients directly before (Pre), directly after (Post) and on the following day (1d) of PCI. Data are presented as mean ± SEM; in case of SAP, n = 21, and for ACS, n = 20. Significant intragroup differences: Pre/1d, Post/1d; p* <0.05, p** <0.01, p**** ≤0.0001; significant intergroup differences (SAP vs ACS) for ACTH: 1d, px** = p** <0.01; for cortisol: Pre, p* <0.05.
Figure 2.
Flow cytometry analysis of granulocytes in CAD patients undergoing PCI.
Percentages of marker-bearing granulocytes and mean fluorescence intensities (MFI) of granulocytes reflect the cell surface appearance of activation markers directly before, directly after and on the following day of PCI (Pre, Post, 1d) in patients with stable angina pectoris (SAP) or with acute coronary syndrome (ACS). Data are mean ± SEM; in case of SAP, n = 21, and for ACS, n = 20 (ACS exception: 1d values, n = 19). Significant differences: p* <0.05, p** <0.01, p*** <0.001, p**** ≤0.0001.
Figure 3.
Plasma levels of lactoferrin and IL-6 in CAD patients undergoing PCI.
Blood samples were collected from stable angina pectoris (SAP) and from acute coronary syndrome (ACS) patients directly before (Pre), directly after (Post) and on the following day (1d) of PCI. Data are presented as mean ± SEM; in case of SAP, n = 21 (IL-6: n = 18), and for ACS, n = 20 (IL-6: n = 19, except: Post: n = 18). Significant differences: p*** <0.001, p**** ≤0.0001.