Table 1.
Demographic information for the patient sample and control sample.
Table 2.
The peak latencies and amplitudes of N1, P2, MMN and LDN at Fz between tinnitus before or after rTMS and control group.
Figure 1.
The THI score in post-treatment as compared to themselves before treatment (from THI 68±10 pre-treatment decrease to 32±6 post-treatment, P<0.01).
Figure 2.
The ERP waveforms and brain topographic maps of tinnitus patients before rTMS.
N1, P2 and MMN are denoted here, but no obvious LDN is present in the ERP results of this group.
Figure 3.
The ERP waveforms and brain topographic maps oftinnitus patients after rTMS.
N1, P2, MMN and LDN are denoted in this figure, and the amplitudes and latencies of N1 and MMN are different from those by normal controls and tinnitus patients before rTMS.
Figure 4.
The ERP waveforms and brain topographic maps of normal control group.
N1, P2 and two contrast waveforms between stimuli conditions MMN and LDN are denoted in this figure along with their brain topographic maps below.