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Figure 1.

Laser capture microdissection of nodule cells.

Panels represent 8 µM thick longitudinal sections of 3-week old Medicago nodules before capture (a,d,g,j,m), after capture (b,e,h,k,n) and captured/isolated cells (c,f,i,l,o). Cells/tissues were isolated from the meristem (m; a–c), distal infection zone (diz; d–f), proximal infection zone(piz; g–i), infected cells (ic; j–l) and uninfected cells (uic; m–o) from the fixation zone.

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Figure 1 Expand

Figure 2.

LCM data validation.

(a,b) In situ localization of MtENOD12 (antisense probe) in the infection zone of longitudinal sections of 14-day-old Medicago nodules, representing brightfield (a; signal appears as black dots) and epipolarization images (b). (c,d) Promoter-GUS analysis of Medicago ROP GTPase (Mtr.35940.1.S1_at, Mtr.15539.1.S1_at), showing β-glucoronidase (GUS) activity in the nodule meristem. (e,f) Promoter-GUS analysis of MtENOD8.2, showing β-glucoronidase activity in the non-infected cells of the nodule as well as in the nodule parenchyma.

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Figure 2 Expand

Table 1.

Number of genes showing cell/tissue specific or enriched expression.

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Table 1 Expand

Table 2.

Selection of genes with known expression profiles used for validation.

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Table 2 Expand