Figure 1.
Amplitude spectra of one patient (gradiometers; eyes open).
Three channels from both hemispheres, showing peaks of rolandic ∼10-Hz, rolandic beta, and occipital alpha activity. Rolandic ∼10-Hz oscillations are clearly increased in the affected hemisphere (AH). UH; unaffected hemisphere. T0, 1 week; T1, 1month; T2, 3 months after stroke.
Table 1.
Clinical details of the patients.
Table 2.
Frequencies (Hz) and amplitudes of spectral peaks in the patients (fT/cm√Hz) over the temporo-parietal (eyes open) and occipital (eyes closed) areas.
Figure 2.
Source localizations of ∼10-Hz oscillations in patients with left hemispheric stroke and in control subjects.
Averages of source localizations of ∼10-Hz oscillations (arbitrary scale) estimated with fdMCE in the patients with left hemispheric stroke (eyes-open/eyes-closed) and in the control subjects (eyes-open).
Figure 3.
Mean (+SEM) source strength of ∼10-Hz oscillations over the temporo–parietal region (eyes open/eyes closed).
AH, affected hemisphere. UH, unaffected hemisphere. T0, 1 week; T1, 1month; T2, 3 months after stroke. *p<0.05.
Figure 4.
Spectrum and time-frequency representation of ∼10-Hz oscillations from one channel over the temporo–parietal region in one patient (eyes open).
The amplitude spectrum of the channel is shown on the left (spectral density in arbitrary units). The burst-like nature of ∼10-Hz oscillations is illustrated in the time-frequency representation on the right.
Table 3.
Source strength estimates (nAm) of ∼10-Hz activity over the temporo-parietal region in all patients (mean ± SEM), and low-frequency (∼1-Hz) oscillations in the patients, in whom low-frequency oscillations were detected (mean ± SEM).
Figure 5.
Spectrum and source localization of abnormal low-frequency magnetic activity (ALFMA).
A) Amplitude spectra (eyes closed) of spontaneous 0–5-Hz brain oscillations in one patient and one control subject. Two channels from the amplitude spectra of spontaneous 0–5-Hz brain oscillations in one patient at T0, T1, and T2 and in one control subject. AH, channel from the perilesional area in the affected hemisphere. UH, channel from the corresponding region in the unaffected hemisphere. T0, 1 week; T1, 1month; T2, 3 months after stroke. B) Source localization of ALFMA in one patient above Source localization of ALFMA (arbitrary scale) projected on the surface of the standard brain volume (same patient as in A). below Source localizations of ALFMA projected on the MRI of the patient. Lesion localization is shown on the left.