Table 1.
Patient characteristics and demographics by study groups.
Table 2.
Independent risk factors associated with BC (case-control analysis).
Table 3.
Incidence of BC in Cryo group and No-Cryo group.
Table 4.
Baseline in No-cryo group versus cryo group.
Table 5.
Independent risk factors associated with BC (cohort analysis).
Figure 1.
Liver biopsies from BC patients.
Representative histopathology images of BC patients liver biopsies, A,C are from No-cryo group and B,D are from Cryo group. In the No-cryo group, the structure of hepatic lobule was integrated, normal hepatic plates were observed, hepatic cells occurred vacuolar degeneration, bile duct epithelium hyperplasia and a few inflammatory cells infiltration, cholestasis was seen (A, C). The integrity of the lobular structure was loss. Intrahepatic bile ducts proliferated significantly, part of the bile duct disappeared. Bile duct epithelial deformation, atrophy, shedding, ?the portal area shows infiltration of lymphocytes, the intrahepatic seen varying degrees of cholestasis, micro-thrombosis was observed in various sized portal area vessels (B, D).
Table 6.
Histopathological Features of Liver Biopsies from 37 BC patients.
Figure 2.
One year BC-free survival curve for live-transplant patients.
One year BC-free survival curve for live-transplant patients according to the Kaplan–Meier method. BC-free survival probability of Patients received intraoperative cryoprecipitate transfusions(red line) was statistically significant lower when compared to the group that received no cryoprecipitate (blue line)(P<0.001).
Table 7.
Analysis of BC occurrence.