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Figure 1.

Field site locations.

Markers indicate names and field site locations in Canterbury, South Island, New Zealand.

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Table 1.

Coordinates for all field sites located on the South Island, New Zealand, including average site temperature (±SE) during the sampling period with rainfall and altitude data.

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Figure 2.

Temperature effects on each trophic level.

Relationship between mean daily temperatures and (A) aphid abundance (Z = 0.18, P = 0.857), (B) proportion of aphids parasitised (Z = 5.91, P<0.001), and (C) proportion of aphids hyperparasitised (Z = 1.71, P = 0.087). Dashed lines indicate non-significant effects. Note: data are site averages, whereas individual measurement dates were analysed, grouped by sites, in the mixed effects models.

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Table 2.

Results for GLMMs for all field data on the relationships between temperature and aphid population growth and parasitism.

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Figure 3.

Interactive effects of temperature and drought on aphid population growth.

Rate of aphid population growth in laboratory mesocosms under 3 treatments, calculated using aphid abundance in parasitoid (Diaeretiella rapae) treatments minus aphid abundances in the predator-free control to give the overall net predator effect (±SE). Temperature caused the greatest reduction in aphid population growth (Z = −4.87, P<0.001) followed by drought (Z = −5.92, P<0.001). In contrast, in the drought × temperature treatment (Z = 11.29, P<0.001) treatment, aphid population growth was positive.

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Figure 4.

Effects of temperature and drought on parasitoid fitness.

(A) Parasitoid longevity under drought, elevated temperature, drought and elevated temperature, and control treatments (±SE). (B) Number of offspring per female (dark bars) and percentage of successful adult emergence (light bars) (*temperature: Z = 2.29, P = 0.02, **drought × temperature: Z = −3.26, P = 0.001).

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Table 3.

Results for all laboratory experiments on the effects of elevated temperature, drought, and their interaction, on parasitoid life-history parameters and aphid population response to parasitism using GLMMs, GLMs, and two-way ANOVAs.

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