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Figure 1.

Cytokine secretion profiling of islet-antigen-specific T cells by the Direct assay in healthy adults.

PBMCs obtained from ten healthy adults (HS#1–10) were cultured for 48 h with GAD65, ZnT8, and PPI 15-mer peptide clusters (GAD65 C1-C14, ZnT8 C1-C16, and PPI C1-C3). Amounts of IL-2, IL-10, IL-13, IL-17A, and IP-10 in the supernatants were measured by a multiplex bead-based cytokine assay. Cytokine data were transformed into heat-map format indicating the fold increase from the background. Blue indicates the strongest cytokine secretion, whereas yellow indicates the background.

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Figure 1 Expand

Table 1.

Numbers of peptides and peptide clusters.

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Table 1 Expand

Figure 2.

Identification of the epitopes in the Direct assay.

CFSE-labeled PBMCs were cultured with single peptides from a “positive” peptide cluster for 8 days. Secreted cytokines were measured on day 2. CD4+/CD8+ T cell proliferation was assessed on day 8 based on the CFSE dilution. (A) Identification of GAD65-specific T cell epitope in HS#4. GAD65 p#73 from peptide cluster #8 (C#8) was found to induce IP-10 secretion. (B) Assessment of CD4+ T cell proliferation in response to GAD65 p#73 in HS#4. (C) Identification of ZnT8-specific T cell epitopes in HS#2. ZnT8 p#2, p#18, p#33, and p#93 were found to induce IP-10 secretion. (D) Assessment of CD4+ T cell proliferation in response to ZnT8 p#2, p#18, p#33, and p#93 in HS#2. (E) Cytokine secretion and T cell proliferation data in the experiments shown in (D) are shown in a heat-map format.

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Table 2.

Summary of the islet-antigen-specific T cell responses in the Direct assay.

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Figure 3.

Detection of ZnT8-specific Th1 cells by intracytoplasmic cytokine expression assay.

PBMCs were stimulated with the identified peptides in the presence of anti-CD28/CD49d antibody and brefeldin A. After 6 h stimulation, the expression of intracytoplasmic cytokines was analyzed by flow cytometry. Gated to CD3+CD4+ cells. (A) Expression of IFN-γ and IL-2 in CD4+ T cells (right) and IFN-γ in CD4+CD45RO+ memory T cells (left) specific for ZnT8 peptides in HS#4. (B) Expression of IFN-γ and IL-2 in CD4+ T cells (right) and IFN-γ in CD4+CD45RO+ memory T cells (left) specific for ZnT8 epitopes in HS#7.

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Figure 4.

Cytokine secretion profiling of specific CD4+ T cells by the Cytokine-driven assay.

(A) Cytokine secretion by ZnT8 p65-specific T cells in HS#4. Results from the Direct assay. (B) ZnT8 p65-specific CD4+ T cell proliferation in HS#4. (C–D) The Cytokine-driven assay. CFSE-labeled PBMCs from HS#4 were cultured with single peptides from ZnT8 C#11 for 7 days in the presence of IL-2 (day 2–7). (C) Cytokine secretion during 24 h re-stimulation with peptides. (D) ICS assay after 6 h re-stimulation in the presence of brefeldin A. Gated to CFSE-diluted CD4+ T cell populations. (E-F) Cytokine secretion profiling of islet-antigen-specific CD4+ T cells in healthy adults by the Cytokine-driven assay. (E) Cytokine secretion during 24 h re-stimulation with peptides. (F) ICS assay after 6 h re-stimulation in the presence of brefeldin A.

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Table 3.

Summary of cytokine secretion profiles in the Cytokine-driven assay.

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Figure 5.

Islet-antigen-specific T cells identified by the Direct assay display higher avidities.

(A) Left: The results of the Direct assay and the Cytokine-driven assay with ZnT8 single peptides from C#11 in HS#7. Right: PBMCs were cultured with titrated concentrations of the indicated peptides (0.04–25 µM) in the presence of IL-2 (days 2–7), and re-stimulated cells at a constant concentration (25 µM). IFN-γ secretion after re-stimulation. (B) Left: The results of the Direct assay and the Cytokine-driven assay with GAD65 single peptides from C#10 in HS#2 (top) and with ZnT8 single peptides from C#11. Right: PBMCs were cultured with titrated concentrations of the indicated peptides in the presence of IL-2 (days 2–7), and re-stimulated cells with 25 µM peptide. IL-13 secretion after re-stimulation.

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Table 4.

Clinical characteristics of T1D patients and HLA-matched controls.

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Table 4 Expand

Figure 6.

Cytokine secretions from ZnT8-specific T cells in T1D patients and healthy adults in the Direct assay.

PBMCs from 15 T1D patients and 15 age/gender/HLA-matched controls were stimulated with 2.5 µM of ZnT8 single peptides (p#2, p#18, p#65, and p#68) in duplicates for 48 hours and six cytokines secreted during the stimulation were measured. (A) Representative results of ZnT8-specific IL-10 and IP-10 secretion with T1D patient samples. (B) Summary of cytokine secretion patterns. The cultures that scored positive in any cytokines were selected. Data was transformed into a heat-map format indicating the fold increase from the background.

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Table 5.

Number of positive cytokine responses induced by 2.5 µM of ZnT8 peptides.

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Figure 7.

Distinct cytokine expression patterns of ZnT8 specific CD4+ T cells between T1D patients and healthy adults.

PBMCs from 15 T1D patients and 15 age/gender/HLA-matched controls were cultured with ZnT8 single peptides for 7 days in the presence of IL-2 (day 2–7). ICS assay was performed after 6 h re-stimulation with the same peptides. Gated to CD4+ T cells. (A) Representative results of cytokine expressions by CD4+ T cells specific for ZnT8 single peptides in T1D patients and controls. (B, C) Cytokine expression patterns of ZnT8-specific CD4+ T cells. 24 from 60 control PBMC cultures (15 subjects×4 peptides) and 22 from 60 T1D PBMC cultures, which contained CD4+ T cells expressing any of three cytokines at a frequency of >0.5%, were used for the analysis. Statistical significance between control group and T1D group was tested by Student’s t-test. (D) Composition of ZnT8-specific CD4+ T cells with distinct cytokine-expression pattern in T1D patients and controls.

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