Table 1.
Characteristics of all study participants and separated for subjects with (AMS+) and without AMS (AMS−) during the passive (PHE) and active hypoxic exposure (AHE).
Table 2.
Oxygen saturation (SpO2) in %, heart rate (HR) in beats per min, systolic (Sys. Bp) and diastolic (Dia. Bp) blood pressure in mmHg of baseline measurements and during hypoxia.
Figure 1.
Regional cerebral changes during the passive hypoxic exposure (PHE) for the entire group (N = 20).
Areas with significant increases of gray (GM) and white matter (WM) volume are presented in yellow, ADC increases are presented in red (p<0.001, uncorrected). Right hemisphere in the figure denotes left hemisphere of the brain and vice versa. A general T1 image provided by xjView 8 was used as background.
Table 3.
Anatomical regions with significant gray (GM) and white matter (WM) volume increases during the passive hypoxic exposure (PHE) (p<0.001, uncorrected).
Table 4.
Anatomical regions with significant increases of apparent diffusion coefficient (ADC) during the passive hypoxic exposure (PHE) (p<0.001, uncorrected).
Figure 2.
Regional cerebral changes during the active hypoxic exposure (AHE) for the entire group (N = 20).
Areas with significant increases of gray (GM) and white matter (WM) volume are presented in yellow, ADC increases are presented in red (p<0.001, uncorrected). Right hemisphere in the figure denotes left hemisphere of the brain and vice versa. A general T1 image provided by xjView 8 was used as background.
Table 5.
Anatomical regions with significant gray (GM) and white matter (WM) volume increases during the active hypoxic exposure (AHE) (p<0.001, uncorrected).
Table 6.
Anatomical regions with significant increases of apparent diffusion coefficient (ADC) during the active hypoxic exposure (AHE) (p<0.001, uncorrected).