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Figure 1.

The biosynthetic pathways for thiamine and histidine in S. enterica.

Panel A shows relevant steps from the thiamine biosynthetic pathway while panel B shows relevant steps in the histidine synthetic pathway, both in S. enterica. The enzymatic steps that lead to the formation of their respective end products are show. Abbreviations: Gln, glutamine; PRPP, phosphoribosyl pyrophosphate; PRA, phosphoribosylamine; Gly, glycine; GAR, glycineamide ribonucleotide; ProFAR, 1-(5-phosphoribosyl)-5-[(5-phosphoribosylamino)methylideneamino]imidazole-4-carboxamide; PRFAR, 5-[(5-Phospho-1-deoxyribulos-1-ylamino)methylideneamino]-1-(5-phosphoribosyl)imidazole-4-carboxamide.

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Figure 1 Expand

Table 1.

Bacterial strains.

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Table 1 Expand

Table 2.

Diverse mutations in hisA allow PurF-independent PRA synthesis.

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Table 2 Expand

Figure 2.

Growth analysis of hisA mutant strain.

Growth curves were performed by monitoring optical density (OD) at 650 nm. Shown is a purF2085 gnd174::MudJ hisA1451 (DM10350) strain grown in minimal glucose adenine medium (open triangle), with thiamine (open square), with histidine (filled triangle), and with histidine and thiamine (filled square).

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Figure 2 Expand

Table 3.

Growth rates of some hisA mutant strains are increased by exogenous histidine.

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Table 3 Expand

Table 4.

Metabolic flux to ProFAR is required for PurF-independent PRA synthesis.

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Figure 3.

HPLC separation of ProFAR breakdown products.

The dashed line indicates the trace of stock 1 mM ProFAR used for this assay. The solid line (offset) indicates the trace of 1 mM ProFAR pH 7.5 after incubation at 37°C for 26 hours. Stars indicate unknown break down products. Abbreviations: AICAR, 5-amino-4-imidazolecarboxamide ribonucleotide; ProFAR, 1-(5-phosphoribosyl)-5-[(5-phosphoribosylamino)methylideneamino]imidazole-4-carboxamide.

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Figure 3 Expand

Figure 4.

Possible mechanisms for PRA formation from ProFAR.

General mechanisms for PRA formation from ProFAR are depicted schematically. In pathway I, ProFAR is hydrolyzed to generate R5P by a mechanism that likely requires an enzyme. Ammonia is also released from the non-R5P product and is then available for non-enzymatic formation of PRA. It is possible the R5P and/or the ammonia do not leave the active site of the relevant enzyme. Pathway II depicts the formation of PRA as a direct product and implicates an undefined enzyme-catalyzed mechanism. Abbreviations: ProFAR, 1-(5-phosphoribosyl)-5-[(5-phosphoribosylamino) methylideneamino] imidazole-4-carboxamide; R5P, ribose-5′-phosphate; PRA, phosphoribosylamine.

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Figure 4 Expand