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Figure 1.

Experimental model.

The horizontal arrows show the Chronic Low Frequency Stimulation period 1 hour (A) or 24 hours (B) after recovering from the surgery (4 days). Then both stimulated and non-stimulated contralateral tibialis anterior muscles were sampled at 0 hours (right after CLFS), 1, 6, 12 and 24 hours of rest. Collection of the samples was done always at the same hour of the day for both protocols.

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Figure 2.

Glycogen content and the absorbance branching index.

(A) Glycogen content of 1 hour (▪ bar) or 24 hours (▪ bar) stimulated muscles (µmols of glucose/g tissue). (B) Wavelength of the maximum absorbance peak (nm) as branching index (▪ dots are 1 hour CLFS samples and ▪ dots are 24 hours CLFS samples). Control group (□ bar or dot) was composed with the pool of all contralateral non-stimulated muscle samples results (n = 50). n = 5 for the stimulated and rested groups. (C) The same amount (2.4 micrograms) of control (white circles), poorly branched normal content (24 h CLFS+1 h rest, grey triangles), poorly branched supercompensated (24 h CLFS+6 h rest, grey diamond) and normal branched supercompensated (black squares, 24 h CLFS+24 h rest) purified glycogen samples were elongated using GPh-b. Glucose incorporation from G1P was monitored at 5, 15, 30, 4 and 60 minutes. n = 3–5. Data are means ± SE. Bars or dots with the same letter are not significantly different from each other. *: p<0.05 compared with control.

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Table 1.

Blood parameters, metabolites and nucleotide levels in muscle.

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Table 1 Expand

Figure 3.

HK2 cDNA change.

Fold change in hk2 mRNA content of 1 hour (▪) or 24 hours (▪) stimulated samples compared to control situation measured with Real-Time PCR. Control group (□) was composed with the pool of all contralateral non-stimulated muscle samples results (n = 50). n = 5 for all the other groups. Data are means ± SE. Bars with the same letter are not significantly different from each other.

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Table 2.

Enzyme activities in skeletal muscle.

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Table 2 Expand

Figure 4.

Change of glycogenin-1 protein content.

Fold change in glycogenin normalized by total protein content of 1 hour (▪) or 24 hours (▪) stimulated samples compared to control situation (□) measured by Western-blot of Glycogenin-1. The inset shows a representative blot for each situation (from 0 to 24 hours of rest): in the upper panel samples with 1 hour of CLFS, and the lower panel the groups with 24 hours of CLFS. C is the contralateral non-stimulated control sample of each sample showed. In the bar chart the control group was composed with the pool of all contralateral1 non-stimulated muscle samples results (n = 50). n = 5 for the stimulated and rested groups. Data are means ± SE. There was no statistical difference under any condition.

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Figure 5.

Total mGS protein and phosphorylation content of the different sites.

Fold change of 1 hour (▪) or 24 hours (▪) stimulated samples compared to control situation (□) measured by Western-blot of: (A) Total mGS protein content normalized by total protein and phosphorylation of mGS in (B) site 1a, (C) site 1b, (D) site 2, (E) site 2+2a, (F) site 3a and (G) site 3b, normalized by mGS. The control was composed with the pool of all contralateral non-stimulated muscle samples results (n = 50). For the stimulated and rested groups n = 5. The upper panels of each chart contain representative blots to the total mGS or the phosphorylated sites: 1 hour CLFS the upper lane and 24 hours CLFS the lower lane. C: The correspondent non-stimulated control, and 0 h, 1 h, 6 h, 12 h and 24 h are the time of rest after the stimulation. Data are expressed as box and whisker plot with the median as a transversal line in the box. Groups with the same letter are not significantly different from each other.

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Figure 6.

The activation state of AMPK.

(A) Representative blots of AMPK-P (T172), AMPKα1 and AMPKα2 isoforms. 1 hour CLFS samples on the left and 24 hours CLFS on the right. Samples are C: The correspondent non-stimulated control, and 0 h, 1 h, 6 h, 12 h and 24 h are the time of rest after the stimulation. (B) Quantification of the AMPK-P (T172) Western-blot blot normalized by AMPKα1 in fold change (from 0 to 160 fold) compared to control and in a different scale (C) with a break from 7 to 35 to better display the data in the control samples and samples rested from 6 to 24 hours. Quantification of AMPKα1 (D) and AMPKα2 (E) in fold change compared to control from the Western-blot normalized by total protein content. 1 hour (▪) and 24 hours (▪) stimulated samples n = 5. Control group (□) was composed with the pool of all contralateral non-stimulated muscle samples results (n = 50). Data are means ± SE. Bars with the same letter are not significantly different from each other.

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