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Figure 1.

Chronic stress regulates BDNF expression in CA3 and BLA in contrasting manner.

Data are presented as mean ± SEM. A Experimental design of chronic stress (CIS+1d) followed by micro dissection of area CA3 from transverse hippocampal sections or BLA from coronal sections. B BDNF expression in area CA3 is decreased after chronic stress (Control, n = 5; CIS+1d, n = 5), C Chronic stress up regulates BDNF expression in BLA (Control, n = 6; CIS+1d, n = 8). Chronic stress induced changes in BDNF expression profile is normalized to respective control level. D Serum corticosterone levels are up regulated after chronic stress in comparison to controls (Control, n = 5; CIS+1d, n = 6). Significant differences *, p<0.05; Unpaired, two-tailed, Student's t-test was used to compare stress and control groups.

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Figure 2.

Contrasting profiles of BDNF in CA3 and BLA in response to recovery from chronic stress.

Data are presented as mean ± SEM A Experimental design of 21 days of recovery from chronic stress paradigm (CIS+21d). B BDNF expression in area CA3 comes back to control levels post recovery from chronic stress (Control, n = 6; CIS+21d, n = 6), C Chronic stress induced up regulation in BDNF expression in BLA persists even after twenty one days of recovery (Control, n = 4; CIS+21d, n = 6). BDNF expression profile is normalized to respective control level. D The up regulation of serum corticosterone levels persists even after recovery from chronic stress (Control, n = 7; CIS+21d, n = 6). Significant differences *, p<0.05; Unpaired, two-tailed, Student's t-test was used to compare stress and control groups.

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Figure 3.

Acute stress modulates BDNF profile differentially in CA3 and BLA.

Data are presented as mean ± SEM. A Experimental design of acute stress (AIS+1d) and recovery for 10 days from acute stress (AIS+10d). B BDNF expression in area CA3 in response to acute stress. Acute stress does not change the BDNF levels in CA3 (Control, n = 9; AIS+1d, n = 9; AIS+10d, n = 6, F(2,21) = 2.11, p>0.05). C Acute stress up regulates BDNF expression in BLA and this up regulation persists even after recovery (Control, n = 6; AIS+1d, n = 5; AIS+10d, n = 6, F(2,14) = 4.82, p<0.05). BDNF expression profile was normalized to respective control levels and evaluated by Fisher's post-hoc. D Effect of acute stress and recovery from acute stress on serum corticosterone levels (Control, n = 11; AIS+1d, n = 5; AIS+10d, n = 6, Kruskal-Wallis ANOVA, χ2(2) = 10.01, p<0.05, Mann-Whitney post-hoc, p<0.05). Significant differences *, p<0.05.

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Figure 3 Expand

Table 1.

Summary of body weight measures.

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Table 1 Expand