Figure 1.
T1-weighted MRI scans of the patient.
Consecutive sequences of axial T1-weighted MRI scan of the patient's brain three years after her strokes (right hemisphere is on the left side of the picture). Hypodense lesion massively extends from the bilateral superior temporal gyrus toward the transverse temporal gyrus, the pulvinar, thalamus and insula and the middle temporal gyrus. The right hemisphere was further damaged in the lentiform nucleus, the postcentral gyrus, the inferior frontal gyrus and the inferior parietal lobe.
Figure 2.
Grand-averaged auditory evoked potentials to sound onsets.
Auditory brainstem responses (ABRs; top), middle-latency auditory evoked potentials (MLAEPs; middle), and long-latency auditory evoked potentials (LLAEPs; bottom) recorded by routine clinical protocols in the patient with cortical deafness (left) and in the control group (right). Note the integrity of brainstem auditory responses and the absence of bilateral middle- and long-latency evoked potentials, characterizing cortical deafness.
Figure 3.
Grand-averaged event related potentials (ERPs) elicited by classic oddball at electrode Cz.
Grand-averaged ERP waveforms at electrode Cz and thirty-one channel ERP topographical maps recorded in the patient (B) and controls (A). The latency is referred at the time point at which the positive peak between 300 and 700 ms latency is maximum at Cz electrode site. ERP waveform from the patient revealed a significantly higher P3 at the left posterior and central sites in response to right stimulation. No P3 could be recorded after left ear stimuli.