Trillin protects against doxorubicin-induced cardiotoxicity through regulating Nrf2/HO-1 signaling pathway
Fig 5
Trillin attenuated DOX-induced oxidative stress.
(A-D) CAT, GSH, SOD, and MDA lvels in cardiac tissue of mice. (E-H) CAT, GSH, SOD, and MDA levels in H9c2 cells (n = 6). ##P < 0.01 vs control; * P < 0.05, **P < 0.01 vs DOX.