Trillin protects against doxorubicin-induced cardiotoxicity through regulating Nrf2/HO-1 signaling pathway
Fig 2
Trillin improved DOX-induced cardiac dysfunction in C57 mice.
(A) Schematic protocol for DOX and trillin treatment. (B-G) Echocardiography parameters of HR, LVEF, LVFS, SV, LVIDs, and LVIDd in mice (n = 8). #P < 0.05, ##P < 0.01 vs control; * P < 0.05, vs DOX.