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SIRT5 deficiency suppresses mitochondrial ATP production and promotes AMPK activation in response to energy stress

Fig 5

Sirt5 KO ameliorates cardiac dysfunction caused by TAC.

(A-F) Left ventricular cardiac functional parameters between male Sirt5 KO mice and WT littermates at 5 weeks after TAC surgery (n = 6–10 animals per group), including left ventricular internal diastolic diameter (LVIDd) (A), left ventricular internal systolic diameter (LVIDs) (B), left ventricular end-diastolic volume (LVEDV) (C), left ventricular end-systolic volume (LVESV) (D), left ventricular ejection fraction LVEF% (E), and left ventricular shortening fraction LVFS% (F). Data are shown as mean ± SD of at least 3 independent experiments, one-way ANOVA. *denotes the P< 0.05 and **denotes the P < 0.01 for the indicated comparisons. n.s. = not significant.

Fig 5

doi: https://doi.org/10.1371/journal.pone.0211796.g005