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Glucuronidation by UGT1A1 Is the Dominant Pathway of the Metabolic Disposition of Belinostat in Liver Cancer Patients

Figure 6

Association between glucuronidation of belinostat and UGT1A1 substrates (A, B, and C).

A: Bilirubin-G; B: thyroxine-4-G/CPT-11; C: SN38-G/CPT11.

Figure 6

doi: https://doi.org/10.1371/journal.pone.0054522.g006