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Expression of human A53T alpha-synuclein without endogenous rat alpha-synuclein fails to elicit Parkinson’s disease-related phenotypes in a novel humanized rat model
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Nicole K. Polinski ,
Roles Methodology, Project administration, Writing – original draft, Writing – review & editing
* E-mail: npolinski@michaeljfox.org
Affiliation Research Programs, The Michael J. Fox Foundation for Parkinson’s Research, New York, New York, United States of America
⨯ - Jukka Puoliväli,
- Leena Rauhala,
- Taina-Kaisa Stenius,
- Timo Bragge,
- Teija Parkkari,
- Anna-Maija Penttinen,
- Yi Chen,
- Omar S. Mabrouk,
- Kelly E. Glajch,
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Warren D. Hirst,
Roles Conceptualization, Funding acquisition, Methodology
Affiliation Neurodegenerative Diseases Research Unit, Biogen, Cambridge, Massachusetts, United States of America
⨯ - Michael Perkinton,
- Terina N. Martinez,
- Mark A. Frasier
https://orcid.org/0000-0003-4613-9115
Expression of human A53T alpha-synuclein without endogenous rat alpha-synuclein fails to elicit Parkinson’s disease-related phenotypes in a novel humanized rat model
- Nicole K. Polinski,
- Jukka Puoliväli,
- Leena Rauhala,
- Taina-Kaisa Stenius,
- Timo Bragge,
- Teija Parkkari,
- Anna-Maija Penttinen,
- Yi Chen,
- Omar S. Mabrouk,
- Kelly E. Glajch
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- Published: August 8, 2025
- https://doi.org/10.1371/journal.pone.0329823