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Early chronic suppression of microglial p38α in a model of Alzheimer’s disease does not significantly alter amyloid-associated neuropathology
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David J. Braun ,
Contributed equally to this work with: David J. Braun, Hilaree N. Frazier
Roles Conceptualization, Funding acquisition, Project administration, Supervision, Writing – original draft, Writing – review & editing
Affiliations Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky, United States of America, Department of Neuroscience, University of Kentucky, Lexington, Kentucky, United States of America
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Hilaree N. Frazier ,
Contributed equally to this work with: David J. Braun, Hilaree N. Frazier
Roles Investigation, Supervision, Writing – original draft, Writing – review & editing
Affiliation Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky, United States of America
⨯ - Verda A. Davis,
- Meggie J. Coleman,
- Colin B. Rogers,
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Linda J. Van Eldik
Roles Conceptualization, Funding acquisition, Project administration, Writing – review & editing
* E-mail: linda.vaneldik@uky.edu
Affiliations Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky, United States of America, Department of Neuroscience, University of Kentucky, Lexington, Kentucky, United States of America
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https://orcid.org/0000-0002-6967-0489
Early chronic suppression of microglial p38α in a model of Alzheimer’s disease does not significantly alter amyloid-associated neuropathology
- David J. Braun,
- Hilaree N. Frazier,
- Verda A. Davis,
- Meggie J. Coleman,
- Colin B. Rogers,
- Linda J. Van Eldik
- Published: May 31, 2023
- https://doi.org/10.1371/journal.pone.0286495