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Open Access

Peer-reviewed

Research Article

LC3B is not recruited along with the autophagy elongation complex (ATG5-12/16L1) at HCV replication site and is dispensable for viral replication

Ahmed M. Fahmy,

Roles Conceptualization, Investigation, Writing – original draft

Affiliations INRS-Institut Armand-Frappier, Institut National de la Recherche Scientifique, Laval, Canada, Molecular Medicine Research Group, Department of Reproductive Medicine, Division of Medical Research, National Research Centre (NRC), Dokki, Egypt
⨯
Marwa Khabir,

Roles Investigation, Methodology

Affiliation INRS-Institut Armand-Frappier, Institut National de la Recherche Scientifique, Laval, Canada
⨯
Matthieu Blanchet,

Roles Supervision

Affiliation INRS-Institut Armand-Frappier, Institut National de la Recherche Scientifique, Laval, Canada
⨯
Patrick Labonté

Roles Conceptualization, Project administration, Supervision, Writing – review & editing

* E-mail: patrick.labonte@iaf.inrs.ca

Affiliation INRS-Institut Armand-Frappier, Institut National de la Recherche Scientifique, Laval, Canada

http://orcid.org/0000-0001-7262-3125

⨯

LC3B is not recruited along with the autophagy elongation complex (ATG5-12/16L1) at HCV replication site and is dispensable for viral replication

Ahmed M. Fahmy,
Marwa Khabir,
Matthieu Blanchet,
Patrick Labonté

x

Published: October 4, 2018
https://doi.org/10.1371/journal.pone.0205189

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Autophagic cell death
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Transfection
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Viral replication complex
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Viral replication
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Hepatitis C virus
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Antibodies
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Protein interactions
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RNA structure
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