Structural instability of IκB kinase β promotes autophagic degradation through enhancement of Keap1 binding

• Mayu Kanamoto,

  Roles Formal analysis, Investigation, Writing – original draft

  Affiliations Department of Molecular Medical Science, Graduate School of Biomedical Science, Hiroshima University, Hiroshima, Japan, Department of Dermatology, Mazuda Hospital, Hiroshima, Japan

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• Yoshihiro Tsuchiya,

  Roles Data curation, Funding acquisition, Investigation

  Affiliations Department of Molecular Medical Science, Graduate School of Biomedical Science, Hiroshima University, Hiroshima, Japan, Department of Nutrition and Health Promotion, Hiroshima Jogakuin University, Hiroshima, Japan

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• Yuki Nakao,

  Roles Investigation

  Affiliation Department of Molecular Medical Science, Graduate School of Biomedical Science, Hiroshima University, Hiroshima, Japan

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• Takafumi Suzuki,

  Roles Resources

  Affiliation Department of Medical Biochemistry, Tohoku University Graduate School of Medicine, Sendai, Japan

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• Hozumi Motohashi,

  Roles Resources

  Affiliation Department of Gene Expression Regulation, Institute of Development, Aging and Cancer, Tohoku University, Sendai, Japan

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• Masayuki Yamamoto,

  Roles Resources

  Affiliation Department of Medical Biochemistry, Tohoku University Graduate School of Medicine, Sendai, Japan

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• Hideaki Kamata

  Roles Conceptualization, Data curation, Funding acquisition, Investigation, Methodology, Project administration, Supervision, Writing – original draft, Writing – review & editing

  * E-mail: hkamata@hiroshima-u.ac.jp

  Affiliation Department of Molecular Medical Science, Graduate School of Biomedical Science, Hiroshima University, Hiroshima, Japan

  http://orcid.org/0000-0002-2566-2452

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