Dysfunctional GPR40/FFAR1 signaling exacerbates pain behavior in mice

• Kazuo Nakamoto,

  Affiliation Department of Clinical Pharmacy, School of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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• Fuka Aizawa,

  Affiliation Department of Clinical Pharmacy, School of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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• Kei Miyagi,

  Affiliation Department of Clinical Pharmacy, School of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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• Takuya Yamashita,

  Affiliation Biochemical Toxicology Laboratory, Faculty of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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• Mitsumasa Mankura,

  Affiliation Faculty of Food Culture, Kurashiki Sakuyo University, Kurashiki, Okayama, Japan

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• Yutaka Koyama,

  Affiliation Laboratory of Pharmacology, Faculty of Pharmacy, Osaka Ohtani University, Tondabayashi, Osaka, Japan

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• Fumiyo Kasuya,

  Affiliation Biochemical Toxicology Laboratory, Faculty of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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• Akira Hirasawa,

  Affiliation Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University, Kyoto, Japan

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• Takashi Kurihara,

  Affiliation Department of Pharmacology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan

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• Atsuro Miyata,

  Affiliation Department of Pharmacology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan

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• Shogo Tokuyama

  * E-mail: stoku@pharm.kobegakuin.ac.jp

  Affiliation Department of Clinical Pharmacy, School of Pharmaceutical Sciences, Kobe Gakuin University, Chuo-ku, Kobe, Japan

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