Promoter hypermethylation as a mechanism for Lamin A/C silencing in a subset of neuroblastoma cells

• Ines Rauschert ,

  Contributed equally to this work with: Ines Rauschert, Fabian Aldunate

  Affiliation Laboratory of Cellular Signaling and Nanobiology, Instituto de Investigaciones Biológicas Clemente Estable, Montevideo, Uruguay

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• Fabian Aldunate ,

  Contributed equally to this work with: Ines Rauschert, Fabian Aldunate

  Affiliation Epigenetics of Cancer and Aging Laboratory, Institut Pasteur de Montevideo, Montevideo, Uruguay

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• Jens Preussner,

  Affiliation Bioinformatics Core Unit (BCU), Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany

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• Miguel Arocena-Sutz,

  Affiliation Epigenetics of Cancer and Aging Laboratory, Institut Pasteur de Montevideo, Montevideo, Uruguay

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• Vanina Peraza,

  Affiliation Epigenetics of Cancer and Aging Laboratory, Institut Pasteur de Montevideo, Montevideo, Uruguay

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• Mario Looso,

  Affiliation Bioinformatics Core Unit (BCU), Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany

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• Juan C. Benech ,

  * E-mail: jbenech@iibce.edu.uy (JCB); agrelo@pasteur.edu.uy (RA)

  Affiliation Laboratory of Cellular Signaling and Nanobiology, Instituto de Investigaciones Biológicas Clemente Estable, Montevideo, Uruguay

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• Ruben Agrelo

  * E-mail: jbenech@iibce.edu.uy (JCB); agrelo@pasteur.edu.uy (RA)

  Affiliation Epigenetics of Cancer and Aging Laboratory, Institut Pasteur de Montevideo, Montevideo, Uruguay

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