The abundance of the ARL2 GTPase and its GAP, ELMOD2, at mitochondria are modulated by the fusogenic activity of mitofusins and stressors

Open Access

Peer-reviewed

Research Article

Laura E. Newman ,

Contributed equally to this work with: Laura E. Newman, Cara R. Schiavon

Affiliation Department of Biochemistry, Emory University School of Medicine, Atlanta, Georgia, United States of America
⨯
Cara R. Schiavon ,

Contributed equally to this work with: Laura E. Newman, Cara R. Schiavon

Affiliation Department of Biochemistry, Emory University School of Medicine, Atlanta, Georgia, United States of America
⨯
Chengjing Zhou,

Affiliation Department of Biochemistry, Emory University School of Medicine, Atlanta, Georgia, United States of America
⨯
Richard A. Kahn

* E-mail: rkahn@emory.edu

Affiliation Department of Biochemistry, Emory University School of Medicine, Atlanta, Georgia, United States of America
⨯

Reader Comments (0)

Mitochondria
Is the Subject Area "Mitochondria" applicable to this article?

Thanks for your feedback.
Cell staining
Is the Subject Area "Cell staining" applicable to this article?

Thanks for your feedback.
Glucose
Is the Subject Area "Glucose" applicable to this article?

Thanks for your feedback.
Cell fusion
Is the Subject Area "Cell fusion" applicable to this article?

Thanks for your feedback.
Transfection
Is the Subject Area "Transfection" applicable to this article?

Thanks for your feedback.
Guanosine triphosphatase
Is the Subject Area "Guanosine triphosphatase" applicable to this article?

Thanks for your feedback.
Immunoblotting
Is the Subject Area "Immunoblotting" applicable to this article?

Thanks for your feedback.
Cellular stress responses
Is the Subject Area "Cellular stress responses" applicable to this article?

Thanks for your feedback.