In Vivo Modelling of ATP1A3 G316S-Induced Ataxia in C. elegans Using CRISPR/Cas9-Mediated Homologous Recombination Reveals Dominant Loss of Function Defects

Open Access

Peer-reviewed

Research Article

Altar Sorkaç,

Affiliation Department of Neuroscience, Brown University, Providence, RI, United States of America
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Ivan C. Alcantara,

Affiliation Department of Neuroscience, Brown University, Providence, RI, United States of America
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Anne C. Hart

* E-mail: anne_hart@brown.edu

Affiliation Department of Neuroscience, Brown University, Providence, RI, United States of America

http://orcid.org/0000-0001-7239-4350

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Caenorhabditis elegans
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Heterozygosity
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Alleles
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Mutation
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Adenosine triphosphatase
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Homozygosity
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Genomics
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Homologous recombination
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