Metformin Ameliorates Dysfunctional Traits of Glibenclamide- and Glucose-Induced Insulin Secretion by Suppression of Imposed Overactivity of the Islet Nitric Oxide Synthase-NO System

• Ingmar Lundquist,

  Affiliations Department of Clinical Science, SUS, Division of Islet Cell Physiology University of Lund, Malmö, Sweden, Department of Experimental Medical Science, University of Lund, Lund, Sweden

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• Israa Mohammed Al-Amily,

  Affiliation Department of Clinical Science, SUS, Division of Islet Cell Physiology University of Lund, Malmö, Sweden

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• Sandra Meidute Abaraviciene,

  Affiliations Department of Clinical Science, SUS, Division of Islet Cell Physiology University of Lund, Malmö, Sweden, Dept. of Physiology, Biochemistry, Microbiology and Laboratory Medicine Vilnius University, and Dep. of Regenerative Medicine, State Research Institute Center for Innovative Medicine, Vilnius, Lithuania

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• Albert Salehi

  * E-mail: S_Albert.Salehi@med.lu.se

  Affiliations Department of Clinical Science, SUS, Division of Islet Cell Physiology University of Lund, Malmö, Sweden, Department of Neuroscience and Physiology, Metabolic Research Unit, University of Gothenburg, Gothenburg, Sweden

  http://orcid.org/0000-0001-6120-3539

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