Protection of α-CaMKII from Dephosphorylation by GluN2B Subunit of NMDA Receptor Is Abolished by Mutation of Glu96 or His282 of α-CaMKII

Open Access

Peer-reviewed

Research Article

Protection of α-CaMKII from Dephosphorylation by GluN2B Subunit of NMDA Receptor Is Abolished by Mutation of Glu⁹⁶ or His²⁸² of α-CaMKII

Madhavan Mayadevi ,

Contributed equally to this work with: Madhavan Mayadevi, Kesavan Lakshmi

Affiliation Molecular Neurobiology Division, Rajiv Gandhi Centre for Biotechnology, Thycaud, P. O., Thiruvananthapuram-695014, Kerala, India
⨯
Kesavan Lakshmi ,

Contributed equally to this work with: Madhavan Mayadevi, Kesavan Lakshmi

Affiliation Molecular Neurobiology Division, Rajiv Gandhi Centre for Biotechnology, Thycaud, P. O., Thiruvananthapuram-695014, Kerala, India
⨯
Sudarsana Devi Suma Priya,

Current address: Department of Pharmacology, Yale University School of Medicine, New Haven, Connecticut, United States of America

Affiliation Molecular Neurobiology Division, Rajiv Gandhi Centre for Biotechnology, Thycaud, P. O., Thiruvananthapuram-695014, Kerala, India
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Sebastian John,

Affiliation Molecular Neurobiology Division, Rajiv Gandhi Centre for Biotechnology, Thycaud, P. O., Thiruvananthapuram-695014, Kerala, India
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Ramakrishnapillai V. Omkumar

* E-mail: omkumar@rgcb.res.in

Affiliation Molecular Neurobiology Division, Rajiv Gandhi Centre for Biotechnology, Thycaud, P. O., Thiruvananthapuram-695014, Kerala, India
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