GABA and Endocannabinoids Mediate Depotentiation of Schaffer Collateral Synapses Induced by Stimulation of Temperoammonic Inputs

Open Access

Peer-reviewed

Research Article

Yukitoshi Izumi,

Affiliations Department of Psychiatry, Washington University School of Medicine, St. Louis, MO, United States of America, Taylor Family Institute for Innovative Psychiatric Research, Washington University School of Medicine, St. Louis, MO, United States of America, Washington University Center for Brain Research in Mood Disorders, Washington University School of Medicine, St. Louis, MO, United States of America
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Charles F. Zorumski

* E-mail: zorumskc@wustl.edu

Affiliations Department of Psychiatry, Washington University School of Medicine, St. Louis, MO, United States of America, Taylor Family Institute for Innovative Psychiatric Research, Washington University School of Medicine, St. Louis, MO, United States of America, Washington University Center for Brain Research in Mood Disorders, Washington University School of Medicine, St. Louis, MO, United States of America
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Adenosine
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Hippocampus
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MAPK signaling cascades
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Synapses
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Endocannabinoids
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Glutamate
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Synaptic plasticity
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Gamma-aminobutyric acid
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