Vorinostat Enhances Cytotoxicity of SN-38 and Temozolomide in Ewing Sarcoma Cells and Activates STAT3/AKT/MAPK Pathways

Open Access

Peer-reviewed

Research Article

Valerie B. Sampson,

Affiliation Nemours Center for Cancer and Blood Disorders, Nemours/A.I. duPont Hospital for Children, Wilmington, Delaware, United States of America
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Nancy S. Vetter,

Affiliation Nemours Center for Cancer and Blood Disorders, Nemours/A.I. duPont Hospital for Children, Wilmington, Delaware, United States of America
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Davida F. Kamara,

Affiliation Nemours Center for Cancer and Blood Disorders, Nemours/A.I. duPont Hospital for Children, Wilmington, Delaware, United States of America
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Anderson B. Collier,

Affiliation Department of Pediatrics, Division of Hematology and Oncology, Children's Healthcare of Mississippi, University of Mississippi Medical Center, Jackson, MS, United States of America
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Renee C. Gresh,

Affiliation Nemours Center for Cancer and Blood Disorders, Nemours/A.I. duPont Hospital for Children, Wilmington, Delaware, United States of America
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E. Anders Kolb

* E-mail: eakolb@nemours.org

Affiliation Nemours Center for Cancer and Blood Disorders, Nemours/A.I. duPont Hospital for Children, Wilmington, Delaware, United States of America
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DNA damage
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Drug therapy
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Apoptosis
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DNA repair
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MAPK signaling cascades
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Cancer treatment
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Ewing sarcoma
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Immunoblot analysis
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