Fetal Alcohol Exposure Reduces Dopamine Receptor D2 and Increases Pituitary Weight and Prolactin Production via Epigenetic Mechanisms

Open Access

Peer-reviewed

Research Article

Omkaram Gangisetty,

Affiliation Endocrine Program, Department of Animal Sciences, Rutgers, The State University of New Jersey, New Brunswick, New Jersey 08901, United States of America
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Olivia Wynne,

Affiliation Endocrine Program, Department of Animal Sciences, Rutgers, The State University of New Jersey, New Brunswick, New Jersey 08901, United States of America
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Shaima Jabbar,

Affiliation Endocrine Program, Department of Animal Sciences, Rutgers, The State University of New Jersey, New Brunswick, New Jersey 08901, United States of America
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Cara Nasello,

Affiliation Department of Genetics, Rutgers, The State University of New Jersey, New Brunswick, New Jersey 08901, United States of America
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Dipak K. Sarkar

* E-mail: sarkar@aesop.rutgers.edu

Affiliation Endocrine Program, Department of Animal Sciences, Rutgers, The State University of New Jersey, New Brunswick, New Jersey 08901, United States of America
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Pituitary gland
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DNA methylation
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Alcohol consumption
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Epigenetics
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Estrogens
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Histones
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Gene expression
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Ovariectomy
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