2B4 Is Dispensable for T-Dependent B Cell Immune Responses, but Its Deficiency Leads to Enhanced T-Independent Responses Due to an Increase in Peritoneal Cavity B1b Cells

Open Access

Peer-reviewed

Research Article

Avijit Ray,

Affiliation Blood Research Institute, BloodCenter of Wisconsin, Milwaukee, Wisconsin, United States of America
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Cheng-Yin Yuan,

Affiliation Department of Microbiology and Molecular Genetics, Medical College of Wisconsin, Milwaukee, Wisconsin, United States of America
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Nichole M. Miller,

Affiliation Blood Research Institute, BloodCenter of Wisconsin, Milwaukee, Wisconsin, United States of America
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Hong Mei,

Affiliation Blood Research Institute, BloodCenter of Wisconsin, Milwaukee, Wisconsin, United States of America
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Bonnie N. Dittel

* E-mail: bonnie.dittel@bcw.edu

Affiliations Blood Research Institute, BloodCenter of Wisconsin, Milwaukee, Wisconsin, United States of America, Department of Microbiology and Molecular Genetics, Medical College of Wisconsin, Milwaukee, Wisconsin, United States of America
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B cells
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Immune response
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Spleen
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T cells
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Antibody isotype determination
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Flow cytometry
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NK cells
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Homeostasis
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