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Open Access

Peer-reviewed

Research Article

A Transmembrane Domain GGxxG Motif in CD4 Contributes to Its Lck-Independent Function but Does Not Mediate CD4 Dimerization

Heather L. Parrish,

Affiliation Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯
Caleb R. Glassman,

Affiliation Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯
Madeline M. Keenen,

Affiliation Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯
Neha R. Deshpande,

Affiliations Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America, The Arizona Center on Aging, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯
Matthew P. Bronnimann,

Affiliation Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯
Michael S. Kuhns

* E-mail: mkuhns@u.arizona.edu

Affiliations Department of Immunobiology, The University of Arizona College of Medicine, Tucson, Arizona, United States of America, The Arizona Center on Aging, The University of Arizona College of Medicine, Tucson, Arizona, United States of America, The BIO-5 Institute, The University of Arizona College of Medicine, Tucson, Arizona, United States of America
⨯

A Transmembrane Domain GGxxG Motif in CD4 Contributes to Its Lck-Independent Function but Does Not Mediate CD4 Dimerization

Heather L. Parrish,
Caleb R. Glassman,
Madeline M. Keenen,
Neha R. Deshpande,
Matthew P. Bronnimann,
Michael S. Kuhns

x

Published: July 6, 2015
https://doi.org/10.1371/journal.pone.0132333

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