Oxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain

• Csaba Imre Szalay,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Katalin Erdélyi,

  Affiliation National Institute of Health (NIH/NIAAA/DICBR), Laboratory of Physiological Studies, Section on Oxidative Stress and Tissue Injury, Bethesda, Maryland, United States of America

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• Gábor Kökény,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Enikő Lajtár,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Mária Godó,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Csaba Révész,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Tamás Kaucsár,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Norbert Kiss,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Márta Sárközy,

  Affiliation University of Szeged, Faculty of Medicine, Department of Biochemistry, Szeged, Hungary

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• Tamás Csont,

  Affiliation University of Szeged, Faculty of Medicine, Department of Biochemistry, Szeged, Hungary

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• Tibor Krenács,

  Affiliation 1st Semmelweis University, Department of Pathology and Experimental Cancer Research; MTA-SE Tumor Progression Research Group, Budapest, Hungary

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• Gábor Szénási,

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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• Pál Pacher ,

  Contributed equally to this work with: Pál Pacher, Péter Hamar

  Affiliation National Institute of Health (NIH/NIAAA/DICBR), Laboratory of Physiological Studies, Section on Oxidative Stress and Tissue Injury, Bethesda, Maryland, United States of America

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• Péter Hamar

  Contributed equally to this work with: Pál Pacher, Péter Hamar

  * E-mail: hamar.peter@med.semmelweis-univ.hu

  Affiliation Semmelweis University, Institute of Pathophysiology, Budapest, Hungary

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