Acidosis Mediates the Switching of Gs-PKA and Gi-PKCε Dependence in Prolonged Hyperalgesia Induced by Inflammation

Open Access

Peer-reviewed

Research Article

Acidosis Mediates the Switching of G_s-PKA and G_i-PKC_ε Dependence in Prolonged Hyperalgesia Induced by Inflammation

Wei-Yu Huang,

Affiliation Department of Life Sciences, National Central University, Jhongli, Taiwan
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Shih-Ping Dai,

Affiliation Department of Life Sciences, National Central University, Jhongli, Taiwan
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Yan-Ching Chang,

Affiliation Department of Life Sciences, National Central University, Jhongli, Taiwan
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Wei-Hsin Sun

* E-mail: weihsin@cc.ncu.edu.tw

Affiliations Department of Life Sciences, National Central University, Jhongli, Taiwan, Institute of Systems Biology & Bioinformatics, National Central University, Jhongli, Taiwan
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Hyperalgesia
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Inflammation
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PKA signaling cascade
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Pain
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Acidosis
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Signal inhibition
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Mice
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Protein kinases
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