MARK/Par1 Kinase Is Activated Downstream of NMDA Receptors through a PKA-Dependent Mechanism

Open Access

Peer-reviewed

Research Article

Laura P. Bernard,

Affiliation Department of Neuroscience and Cell Biology, Robert Wood Johnson Medical School, Rutgers, the State University of New Jersey, Piscataway, New Jersey, United States of America
⨯
Huaye Zhang

* E-mail: huaye.zhang@rutgers.edu

Affiliation Department of Neuroscience and Cell Biology, Robert Wood Johnson Medical School, Rutgers, the State University of New Jersey, Piscataway, New Jersey, United States of America
⨯

Reader Comments (0)

Neurons
Is the Subject Area "Neurons" applicable to this article?

Thanks for your feedback.
Hippocampus
Is the Subject Area "Hippocampus" applicable to this article?

Thanks for your feedback.
Receptor antagonist therapy
Is the Subject Area "Receptor antagonist therapy" applicable to this article?

Thanks for your feedback.
Phosphorylation
Is the Subject Area "Phosphorylation" applicable to this article?

Thanks for your feedback.
Synapses
Is the Subject Area "Synapses" applicable to this article?

Thanks for your feedback.
Synaptic plasticity
Is the Subject Area "Synaptic plasticity" applicable to this article?

Thanks for your feedback.
Antibody therapy
Is the Subject Area "Antibody therapy" applicable to this article?

Thanks for your feedback.
Neuronal dendrites
Is the Subject Area "Neuronal dendrites" applicable to this article?

Thanks for your feedback.