Compressive Stress Induces Dephosphorylation of the Myosin Regulatory Light Chain via RhoA Phosphorylation by the Adenylyl Cyclase/Protein Kinase A Signaling Pathway

Open Access

Peer-reviewed

Research Article

Kenji Takemoto,

Affiliation Transdisciplinary Life Science Course, Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan
⨯
Seiichiro Ishihara,

Affiliations Transdisciplinary Life Science Course, Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan, Research Center for Cooperative Projects, Hokkaido University Graduate School of Medicine, Sapporo, Japan
⨯
Takeomi Mizutani,

Affiliation Transdisciplinary Life Science Course, Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan
⨯
Kazushige Kawabata,

Affiliation Transdisciplinary Life Science Course, Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan
⨯
Hisashi Haga

* E-mail: haga@sci.hokudai.ac.jp

Affiliations Transdisciplinary Life Science Course, Faculty of Advanced Life Science, Hokkaido University, Sapporo, Japan, Research Center for Cooperative Projects, Hokkaido University Graduate School of Medicine, Sapporo, Japan
⨯

Reader Comments (0)

Phosphorylation
Is the Subject Area "Phosphorylation" applicable to this article?

Thanks for your feedback.
Mechanical stress
Is the Subject Area "Mechanical stress" applicable to this article?

Thanks for your feedback.
Adenylyl cyclase
Is the Subject Area "Adenylyl cyclase" applicable to this article?

Thanks for your feedback.
Myosins
Is the Subject Area "Myosins" applicable to this article?

Thanks for your feedback.
Actin filaments
Is the Subject Area "Actin filaments" applicable to this article?

Thanks for your feedback.
Actins
Is the Subject Area "Actins" applicable to this article?

Thanks for your feedback.
Silicones
Is the Subject Area "Silicones" applicable to this article?

Thanks for your feedback.
Adenylyl cyclase signaling cascade
Is the Subject Area "Adenylyl cyclase signaling cascade" applicable to this article?

Thanks for your feedback.