Disrupting the Interaction between Retinoblastoma Protein and Raf-1 Leads to Defects in Progenitor Cell Proliferation and Survival during Early Inner Ear Development

Open Access

Peer-reviewed

Research Article

Wenyan Li,

Affiliations Department of Otorhinolaryngology, Affiliated Eye and ENT Hospital of Fudan University, Shanghai, China, Department of Otolaryngology and Program in Neuroscience, Harvard Medical School and Eaton Peabody Laboratory, Massachusetts Eye and Ear Infirmary, Boston, Massachusetts, United States of America
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Shan Sun,

Affiliation Research Center, Affiliated Eye and ENT Hospital of Fudan University, Shanghai, China
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Yan Chen,

Affiliation Research Center, Affiliated Eye and ENT Hospital of Fudan University, Shanghai, China
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Huiqian Yu,

Affiliation Department of Otorhinolaryngology, Affiliated Eye and ENT Hospital of Fudan University, Shanghai, China
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Zheng-Yi Chen,

Affiliation Department of Otolaryngology and Program in Neuroscience, Harvard Medical School and Eaton Peabody Laboratory, Massachusetts Eye and Ear Infirmary, Boston, Massachusetts, United States of America
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Huawei Li

* E-mail: lihuawei63@gmail.com

Affiliations Institute of Stem Cell and Regeneration Medicine, Institutions of Biomedical Science, Fudan University, Shanghai, China, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, China
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Inner ear
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Phosphorylation
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Cell cycle and cell division
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Apoptosis
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Cell proliferation
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Gene expression
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Cell differentiation
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Stem cells
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