FLZ Alleviates the Memory Deficits in Transgenic Mouse Model of Alzheimer’s Disease via Decreasing Beta-Amyloid Production and Tau Hyperphosphorylation

Open Access

Peer-reviewed

Research Article

Xiu-Qi Bao ,

Contributed equally to this work with: Xiu-Qi Bao, Ning Li

Affiliation State Key Laboratory of Bioactive Substance and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
⨯
Ning Li ,

Contributed equally to this work with: Xiu-Qi Bao, Ning Li

Affiliation State Key Laboratory of Bioactive Substance and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
⨯
Tao Wang,

Affiliation Beijing Municipal Corps Hospital of Chinese People’s Armed Police Force, Beijing, China
⨯
Xiang-Chen Kong,

Affiliation School of Medicine, Shenzhen University, Beijing, China
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Wen-Jiao Tai,

Affiliation State Key Laboratory of Bioactive Substance and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
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Hua Sun,

Affiliation State Key Laboratory of Bioactive Substance and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
⨯
Dan Zhang

* E-mail: danzhang@imm.ac.cn

Affiliation State Key Laboratory of Bioactive Substance and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China
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Phosphorylation
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Mouse models
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Hippocampus
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Genetically modified animals
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Alzheimer's disease
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Enzyme-linked immunoassays
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Memory
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