Direct Evidence that Scorpion α-Toxins (Site-3) Modulate Sodium Channel Inactivation by Hindrance of Voltage-Sensor Movements

Open Access

Peer-reviewed

Research Article

Zhongming Ma,

Affiliation Department of Biochemistry and Biophysics, Perelman School of Medicine University of Pennsylvania, Philadelphia, Pennsylvania, United States of America
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Jun Kong,

Affiliation Department of Biochemistry and Biophysics, Perelman School of Medicine University of Pennsylvania, Philadelphia, Pennsylvania, United States of America
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Dalia Gordon,

Affiliation Department of Plant Molecular Biology and Ecology, George S. Wise Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel Aviv, Israel
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Michael Gurevitz,

Affiliation Department of Plant Molecular Biology and Ecology, George S. Wise Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel Aviv, Israel
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Roland G. Kallen

* E-mail: rgk@mail.med.upenn.edu

Affiliations Department of Biochemistry and Biophysics, Perelman School of Medicine University of Pennsylvania, Philadelphia, Pennsylvania, United States of America, Mahoney Institute for Neuroscience, Perelman School of Medicine University of Pennsylvania, Philadelphia, Pennsylvania, United States of America
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Toxins
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Membrane potential
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Cysteine
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Depolarization
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Toxin binding
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Sodium channels
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Hyperpolarization
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Scorpions
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