Persistent Inhibition of ABL Tyrosine Kinase Causes Enhanced Apoptotic Response to TRAIL and Disrupts the Pro-Apoptotic Effect of Chloroquine

Open Access

Peer-reviewed

Research Article

Priya Sridevi,

Affiliation Moores Cancer Center, Division of Hematology-Oncology, Department of Medicine, School of Medicine, University of California San Diego, La Jolla, California, United States of America
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May K. Nhiayi,

Affiliation Moores Cancer Center, Division of Hematology-Oncology, Department of Medicine, School of Medicine, University of California San Diego, La Jolla, California, United States of America
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Ryan L. Setten,

Affiliation Moores Cancer Center, Division of Hematology-Oncology, Department of Medicine, School of Medicine, University of California San Diego, La Jolla, California, United States of America
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Jean Y. J. Wang

* E-mail: jywang@ucsd.edu

Affiliation Moores Cancer Center, Division of Hematology-Oncology, Department of Medicine, School of Medicine, University of California San Diego, La Jolla, California, United States of America
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Apoptosis
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Tyrosine kinases
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HT29 cells
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Lysosomes
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Chloroquine
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Immunoprecipitation
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Drug therapy
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DNA damage
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