The Endogenous Th17 Response in NO₂-Promoted Allergic Airway Disease Is Dispensable for Airway Hyperresponsiveness and Distinct from Th17 Adoptive Transfer

• Rebecca A. Martin,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Jennifer L. Ather,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Rebecca Daggett,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Laura Hoyt,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• John F. Alcorn,

  Affiliation Division of Pulmonology, Department of Pediatrics, Children’s Hospital of Pittsburgh of UPMC, Pittsburgh, Pennsylvania, United States of America

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• Benjamin T. Suratt,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Daniel J. Weiss,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Lennart K. A. Lundblad,

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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• Matthew E. Poynter

  * E-mail: matthew.poynter@med.uvm.edu

  Affiliation Vermont Lung Center, Division of Pulmonary Disease and Critical Care, University of Vermont, Burlington, Vermont, United States of America

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