Serial Passaging of Candida albicans in Systemic Murine Infection Suggests That the Wild Type Strain SC5314 Is Well Adapted to the Murine Kidney

Open Access

Peer-reviewed

Research Article

Anja Lüttich,

Affiliation Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology – Hans Knoell Institute Jena (HKI), Jena, Germany
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Sascha Brunke,

Affiliations Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology – Hans Knoell Institute Jena (HKI), Jena, Germany, Center of Sepsis Control and Care, Jena, Germany
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Bernhard Hube,

Affiliations Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology – Hans Knoell Institute Jena (HKI), Jena, Germany, Center of Sepsis Control and Care, Jena, Germany, Friedrich Schiller University, Jena, Germany
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Ilse D. Jacobsen

* E-mail: Ilse.jacobsen@hki-jena.de

Affiliation Department of Microbial Pathogenicity Mechanisms, Leibniz Institute for Natural Product Research and Infection Biology – Hans Knoell Institute Jena (HKI), Jena, Germany
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Candida albicans
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Kidneys
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Microevolution
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Spleen
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Host cells
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Yeast infections
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Fungal diseases
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Oxidative stress
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