Targeted Inactivation of GPR26 Leads to Hyperphagia and Adiposity by Activating AMPK in the Hypothalamus

Open Access

Peer-reviewed

Research Article

Daohong Chen ,

Contributed equally to this work with: Daohong Chen, Xiaolei Liu

Affiliation Department of Cellular and Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States of America
⨯
Xiaolei Liu ,

Contributed equally to this work with: Daohong Chen, Xiaolei Liu

Affiliations Department of Cellular and Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States of America, Department of Pharmacology, School of Pharmaceutical Sciences Central South University, Changsha, China
⨯
Weiping Zhang,

Affiliation Department of Pathophysiology, Second Military Medical University Shanghai, China
⨯
Yuguang Shi

* E-mail: yus11@psu.edu

Affiliation Department of Cellular and Molecular Physiology, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States of America
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Obesity
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Genetically modified animals
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Hypothalamus
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Adipose tissue
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Bioenergetics
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Mouse models
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Body weight
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Phosphorylation
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