SAHA Decreases HDAC 2 and 4 Levels In Vivo and Improves Molecular Phenotypes in the R6/2 Mouse Model of Huntington's Disease

• Michal Mielcarek,

  Affiliation Department of Medical and Molecular Genetics, King's College London, London, United Kingdom

  ⨯
• Caroline L. Benn,

  Affiliation Neusentis, Pfizer Inc., Cambridge, United Kingdom

  ⨯
• Sophie A. Franklin,

  Affiliation Department of Medical and Molecular Genetics, King's College London, London, United Kingdom

  ⨯
• Donna L. Smith,

  Affiliation Department of Medical and Molecular Genetics, King's College London, London, United Kingdom

  ⨯
• Ben Woodman,

  Current address: Leeds Institute for Molecular Medicine. St. James's University Hospital, University of Leeds, Leeds, United Kingdom

  Affiliation Department of Medical and Molecular Genetics, King's College London, London, United Kingdom

  ⨯
• Paul A. Marks,

  Affiliation Cell Biology Program, Sloan-Kettering Institute for Cancer Research, Memorial Sloan-Kettering Cancer Center, New York, New York, United States of America

  ⨯
• Gillian P. Bates

  * E-mail: gillian.bates@kcl.ac.uk

  Affiliation Department of Medical and Molecular Genetics, King's College London, London, United Kingdom

  ⨯