IL-17RA Signaling Amplifies Antibody-Induced Arthritis

Open Access

Peer-reviewed

Research Article

Christian D. Sadik,

Affiliation Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, United States of America
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Nancy D. Kim,

Affiliation Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, United States of America
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Elena Alekseeva,

Affiliation Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, United States of America
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Andrew D. Luster

* E-mail: aluster@mgh.harvard.edu

Affiliation Center for Immunology and Inflammatory Diseases, Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, United States of America
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Arthritis
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Neutrophils
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Ankles
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Chemokines
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Skeletal joints
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Cartilage
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Mouse models
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Rheumatoid arthritis
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