Exposed Hydrophobic Residues in Human Immunodeficiency Virus Type 1 Vpr Helix-1 Are Important for Cell Cycle Arrest and Cell Death

Open Access

Peer-reviewed

Research Article

R. Anthony Barnitz,

Current address: Department of Pediatric Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, United States of America

Affiliations Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America, Immunology Graduate Group, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America
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Benjamin Chaigne-Delalande,

Affiliation Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America
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Diane L. Bolton,

Current address: ImmunoTechnology Section, Vaccine Research Center, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America

Affiliation Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America
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Michael J. Lenardo

* E-mail: lenardo@nih.gov

Affiliations Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, United States of America, Immunology Graduate Group, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America
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Cell cycle and cell division
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Virions
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HIV-1
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Cell death
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Transfection
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Protein interactions
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293T cells
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DNA-binding proteins
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